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Abstract Details

Pyridostigmine as an Effective Treatment for Atonic Colon in Parkinson’s Disease
Movement Disorders
P13 - Poster Session 13 (8:00 AM-9:00 AM)
5-007
Constipation is one of the most frequent non-motor symptoms of Parkinson’s Disease (PD) and contributes to poor quality of life in this population. Conventional therapies included a combination of dietary changes, fiber supplements and bowel regimen drugs, most of which have been ineffective or require further larger studies. 

Our case highlights the efficacy of pyridostigmine for a PD patient with severe gastrointestinal complications refractory to conventional therapies. 

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This patient is a 77-year-old male diagnosed with levodopa-responsive PD at 61 years old. His first nonmotor gastrointestinal complication was sigmoid volvulus from chronic constipation that occurred at age 66 and treated with sigmoidectomy. Four years later, patient’s constipation progressed in frequency and duration with no bowel movements for up to six days. He started a daily bowel regimen consisting of aloe vera gel capsules three times daily, docusate 100mg three times daily, wheat dextrin twice daily, and magnesium hydroxide as needed. 

Despite three months of this regimen, patient was hospitalized for a bowel obstruction requiring a multi-day colon preparation for a colonoscopy with decompression. He was diagnosed with Ogilvie’s Syndrome (colonic pseudo-obstruction) due to an atonic colon. At discharge, polyethylene glycol 17g twice daily and water enemas were added to his regimen.

Patient remained constipated with no bowel movements for 2-4 days, requiring 20-30 minutes for bowel movements. He was started on pyridostigmine 60mg daily, titrated up to three times daily for the autonomic manifestation of constipation in PD. 

One month after initiation of pyridostigmine, he only required 5-10 minutes and had bowel movements twice daily with Bristol Type 5 stools for the first time since his PD diagnosis. 

Pyridostigmine may be an effective medical management for constipation in PD patients, especially those who have failed conventional therapies. 

Authors/Disclosures
Ann N. Ly, MD (Ann Ly)
PRESENTER
Dr. Ly has nothing to disclose.
Mahrin Rahman, DO Dr. Rahman has nothing to disclose.
David D. Song, MD, PhD (UCR Health Neurosciences) Dr. Song has nothing to disclose.